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Collectively, these data point out that pharmacologic administration of GLP-1 (9-36amide) could have a practical role to enhance restoration from I/R harm by way of GLP-1R-independent mechanisms, whereas GLP-1 (7-36amide) however not GLP-1 (9-36amide) impacts cardiac contractility via GLP-1R signaling . In rats, GLP-1 (7-36amide) and, maybe paradoxically, the GLP-1R antagonist exendin (9–39), dose-dependently trigger leisure of the aorta, probably by way of elevation of cAMP formation and activation of KATP channels . Comparable findings have been reported for GLP-1 remedy of type-1 diabetes however GLP-1 enchancment of endothelial function appears to be related to native GLP-1 and isn’t observed when GLP-1R agonists are in contrast head-to-head with equally glucose reducing medications.
Risks / Benefits
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